Another interesting Nephsap teaching point by a question was an under recognized cause of acquired apparent mineralcoritcoid access.
Liver cirrhotic patients with elevated bilirubins and sickle cell crisis patients who have elevated bilirubins have this happen to them. When total bilirubin levels are very high, it is hypothesized that worsening cholestasis led to greater bile acid inhibition of 11B - HSDH type II in the distal nephron and this allows for K wasting via cortisol mediation and HTN as well.
Case reports have been reported in liver cirrhosis and sickle cell crisis patients?
Wonder if we see this or its noticed in Post BMT - VOD syndrome?
- ► 2017 (52)
- ► 2016 (45)
- ► 2015 (63)
- ► 2014 (95)
- ► 2013 (133)
- ► 2012 (201)
- ► 2011 (370)
- Pediatrics Grand rounds continue , the last one fo...
- Chronic renal allograft dysfunction
- TOPIC DISCUSSION: Hypokalemic Nephropathy
- KRND Needs YOU!
- CMV in solid Organ transplantation!
- CLINICAL CASE 30, ANSWERS AND SUMMARY
- In the NEWS: Angiopoietin like 4 , a glycoprotein ...
- Health Care Law Blog: AMA Issues New Policy To Gui...
- Genetic Nephropathies and Kidney Transplantation
- Thrombotic Thrombocytopenic Purpura
- Medicine for residents: a little step towards cost...
- IN THE NEWS- Article on Online Blogging of Confere...
- IN THE NEWS: DETECTIVE NEPHRON's NEXT VENTURE
- Series of Videos from ASN - on varied topics- Kidn...
- TOPIC DISCUSSION: Anesthetics and Kidney Disease
- Concept Map of Hypomagnesemia
- Quiz 9 Answers
- IN THE NEWS- Meta-analysis in Nephrology
- TOPIC DISCUSSION: Camels and their Kidney!
- Sirolimus- the Positive aspects
- Nephsap review: Fluids Electrolytes
- ▼ December (21)